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Treatment with edaravone, initiated at symptom onset, slows motor decline and decreases SOD1 deposition in ALS mice
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Protein Chemistry Fall 2008
Discovery Studio Visualizer 2.01
Users Folders
Teresa Garrett
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Protein Project Sources
Structure of Cu/Zn superoxide dismutase from the heavy-metal-tolerant yeast Cryptococcus liquefaciens strain N6
Denaturational Stress Induces Formation of Zinc-Deficient Monomers of Cu,Zn Superoxide Dismutase: Implications for Pathogenesis in Amyotrophic Lateral Sclerosis
SOD1 and Amyotrophic Lateral Sclerosis: Mutations and Oligomerization
Metalation of the Amyotrophic Lateral Sclerosis Mutant Glycine 37 to Arginine Superoxide Dismutase (SOD1) Apoprotein Restores its Structural and Dynamical Properties in Solution to those of Metalated Wild-Type SOD1
Amyotrophic Lateral Sclerosis and Structural Defects in Cu,Zn Superoxide Dismutase
Zinc and Copper in the Pathogenesis of Amyotrophic Lateral Sclerosis
Gain of Interaction of ALS-linked G93A Superoxide Dismutase with Cytosolic Malate Dehydrogenase
Inflammatory cytokines increase mitochondrial damage in motoneuronal cells expressing mutant SOD1
The Cu-Zn superoxide dismutase (SOD1) inhibits ERK phosphorylation by muscarinic receptor modulation in rat pituitary GH3 cells
Treatment with edaravone, initiated at symptom onset, slows motor decline and decreases SOD1 deposition in ALS mice
A Multinuclear Copper (I) Cluster Forms the Dimerization Interface in Copper-Loaded Human Copper Chaperone for Superoxide Dismutase
Mechanism of Cu,Zn-Superoxide Dismutase Activation by the Human Metallochaperone hCCS
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Muhammad Haq
Cristian Opazo
Bona Ko
Laura Gruessner
Marina H-L
Grace Tan
Mike Higgins
Tasnim Rahman
smith2004
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